Axiom · Volume I · Pathway Atlas
Four plates. Three converging axes. Mechanism diagrams that move along the signal direction so you can read a cascade in the time it takes to find your coffee. Every claim traces to citation.
Every plate maps a research mechanism the published literature treats as a coherent unit. Hover any node to follow its signal.
No interpretation is invented for marketing. Every claim in this atlas traces to peer-reviewed literature or to a referenced clinical trial.
The compounds catalogued here are research reference standards. None are presented as approved therapeutics.
A separate plate breaks down every line of a Certificate of Analysis — purity reads, mass-spec identity, counter-ion, peptide content.
The Atlas · Index
Each plate maps a research mechanism the published literature treats as a coherent unit. Hover any node to follow its signal. No interpretation is invented for marketing — every claim traces to citation.
GLP-1R · GIPR · AMYR co-agonism
The dual-incretin axis is the most-studied metabolic-pathway pair in modern peptide-pharmacology literature. GLP-1 and GIP — both gut-derived — converge on pancreatic beta cells through complementary receptors. The amyli…
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GLP-1R · GIPR · AMYR · GHSR coupling
Where Plate I maps a single axis, Plate II maps two — the incretin / amylin convergence laid over the ghrelin-receptor pulsatile growth-hormone axis. The two share no obvious endpoint; they share a research-design patter…
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NO · actin · MMP / collagen modulation
Plate III maps the canonical tissue-repair reference trio. BPC-157 and TB-500 act through complementary mechanisms — nitric-oxide / angiogenesis and actin sequestration / cell migration respectively. GHK-Cu, the copper t…
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Six compounds · three converging axes
Plate IV is the integration plate. Six compounds across three research axes — incretin / amylin, ghrelin-receptor / GH, and tissue repair / ECM — drawn as a single intersecting field. Research programs that span multiple…
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GLP-1R · GIPR · AMYR co-agonism
The dual-incretin axis is the most-studied metabolic-pathway pair in modern peptide-pharmacology literature. GLP-1 and GIP — both gut-derived — converge on pancreatic beta cells through complementary receptors. The amylin axis, co-secreted with insulin, reinforces the same satiet…
Research-utility tools
Four utilities — reconstitution, half-life, dilution conversion, and COA literacy — built on the same data the plates reference. Pure utility. No claims, no protocols.
Bacteriostatic-water volume → resulting concentration + U-100 draw.
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Exponential-decay plots with literature-sourced t½ values per peptide.
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Mass ↔ volume ↔ molarity with 10 preset molecular weights.
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Tick what's on a Certificate of Analysis. Live-scored across 5 categories.
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Comparison preview
Each plate covers a different combination of research axes. Use the matrix to find the plate that maps the literature you're already reading.
| Plate | Subtitle | Compounds |
|---|---|---|
Plate I Dual Incretin & Amylin Axis | GLP-1R · GIPR · AMYR co-agonism | 2 |
Plate II Metabolic × GH-Axis Integration | GLP-1R · GIPR · AMYR · GHSR coupling | 3 |
Plate III Tissue Repair × Extracellular Matrix | NO · actin · MMP / collagen modulation | 3 |
Plate IV Multi-System Integration | Six compounds · three converging axes | 6 |
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Ipamorelin · Tirzepatide · Cagrilintide
BPC-157 · TB-500 · GHK-Cu
Tirzepatide · Cagrilintide · Ipamorelin · BPC-157 · TB-500 · GHK-Cu
Common questions
Every plate organized by receptor family. Three converging axes — metabolic, GH, tissue repair — with the literature behind each.
Pathway-by-pathway summary of the peer-reviewed research that underwrites every claim made in this atlas.
Every line on a Certificate of Analysis explained — purity, mass spec, counter-ion, peptide content, endotoxin.
This atlas is editorial. It does not sell, prescribe, or recommend. Where reference standards for catalogued compounds are required, sourcing notes route to independently-published supplier directories.
Axiom Editorial · Volume I